Tetanus is an entrely preventable sporadic neurologic disorder caused by an exotoxin, tetanospasmin, secreted by Clostridium tetani and characterized by increased muscle tone and spasms in persons with inadequate immunizaton status. The diagnosis is mostly clinical and there is no the gold standard for the recogniton of disease. It is fatal if lef untreated and the outcome is beter if recognized and treated early. Full-blown picture of generalized tetanus is hardly a difcult situaton for the recogniton but when it manifests as localized tetanus without features of generalizaton, diagnosis can be missed in busy hospitals. This is especially true for the cases of cephalic tetanus when Bell’s palsy precedes the development of trismus and generalizaton and the history of trauma is not prominent or is overlooked. This communicaton deals with a review of literature on cephalic tetanus and a typical case of cephalic tetanus referred to us as rabies that made full recovery afer 10 days of treatment with diazepam, metronidazole, and stat dose of human tetanus immunoglobulin. The history of injury was trivial which was forgoten and the inital presentaton of facial palsy of lower motor neuron type was taken as Bell’s palsy. Moreover, the laryngeal and pharyngeal spasms were much more prominent and used to be precipitated by trivial stmulaton such as a puf of air or drinking which were taken as aerophobia and hydrophobia leading to the anecdotal diagnosis of rabies. Key words: Aerophobia, Cephalic tetanus, Facial palsy of lower motor neuron type, Rabies.